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Hip Dysplasia

I have had plans to write a major paper on this frequently misunderstood topic for quite sometime. Unfortunately, the need to write about an ever-increasing number of life-threatening health issues has prevented this from happening before now. But as misconceptions go, this is a high-priority item.

Hip dysplasia is not a “genetic malformation of the hip socket”, as commonly thought, but rather a weakness in the ligaments that support the hip joint. This allows the ball of this ball-in-socket joint to bang away at the joint surface, preventing the socket from forming properly. In other words, the instability created by weak supportive ligaments keeps the body from being able to manufacture a deep, smooth hip socket for the ball to fit snuggly into, resulting in the flattening of the acetabulum (hip socket) and a squaring of the femoral head (the ball). The sockets are shallow because of repetitive trauma to the developing bone (micro-fractures), which prevents the weight-bearing surface of the hip joint from forming properly. These changes worsen drastically as the cartilage covering of the joint wears away and bone-on-bone contact occurs, leaving them with the characteristic signs of the osteoarthritis we see in radiographs of affective dogs.

This may sound like a radical idea but there have been veterinary studies that strongly suggest this for years and years, beginning with those done by orthopedic surgeons and radiologists who developed a technique for measuring joint laxity in Labradors. They took 2 sets of X rays of the hip – one with the ball forced deeply into the socket and one with the ball distracted as much as possible, measuring the differential and trying to use those measurements as a predictor hip dysplasia in the individual.

Why would this be valid? Because the degree of laxity tells us how strong or weak the ligaments surrounding the hip joint might be, which would in turn determine the instability of that joint as the dog grows into maturity. Did all dogs with significant joint laxity go on to become dysplastic? No. Some managed to recover when their muscles developed and the joints stabilized. But others developed radiographic signs of dysplasia as late as 2 years of age, long after the hip sockets were fully formed. Where is that “genetic malformation of the hip socket” in these cases? What criteria do we use in evaluating these cases? We look at how well the hips are seated in the socket? What determines that? The tightness of the ligaments and joint capsule does as much as the depth of the socket. Some of the femoral heads are not anywhere near the socket, appearing to be floating in space on the X ray of these crippled dogs. Loose ligaments.

Imagine a large breed dog, such as a Lab or Saint Bernard, with lax hip joint ligaments as they grow and develop. Imagine the stress and strain this would place on the cartilage of the hip socket as that large or giant breed puppy clumsily runs, slips and slides while chasing the ball or walking across the kitchen floor. Now imagine the same scenario in a more sure-footed cat or toy breed.

We were taught in veterinary school that the breeds of dogs over 40 lbs were the most likely to develop hip dysplasia. This has proven to be true, although I have rarely seen cases of dysplasia in cats and small breeds. In stark contrast, nearly all St. Bernards are dysplastic and the malady is incredibly common in numerous large breeds of dogs – the same breeds of dogs that develop other juvenile bone diseases, including OCD of the shoulder and ununited anconeal or coronoid processes of the elbow. Think there is some stress on the elbows of a rambunctious German Shepherd or Rottie puppy?

All of these maladies are failure of collagen, the building block of our entire skeletal system, which serves as elastic in our connective tissue. Collagen makes up approximately 30% of the protein in our body and is the protein matrix for ligaments, tendons, cartilage, bones, organs, and even blood vessels. Failure of collagen to form properly can have catastrophic results, including skeletal abnormalities, polycystic organ syndromes (e.g. liver, kidneys), and severe vascular syndromes (e.g. aortic aneurysm). A quick study of Ehlers-Danlos Syndrome and Marfan Syndrome tells us most of what we need to know. These run in families just as most collagen disorders (and cancer) run in breeds of dogs. There are “genetics” involved but the truth is much more than skin deep. Food intolerance also runs through families, with 40% of first degree relatives born to a celiac developing the condition. Interestingly, we were taught that if two OFA certified, hip dysplasia-free dogs were bred together, up to 40% of their litter – this exact same percentage – could be born with dysplasia. This is not a coincidence.

The first clue of how to combat this epidemic comes when we grasp the importance of nutrition in the development of our entire body. One of the most important components of collagen is ascorbic acid – good old vitamin C. Without adequate levels of this essential vitamin, we develop scurvy – a horrible condition marked by bleeding gums, open sores, pathological fractures and death if not treated properly. This follows a combination of collagen and immune failure, all resulting from inadequate vitamin C intake.

In the human, where is the vitamin C absorbed? It is absorbed by the small intestine where most nutrients are picked up by the delicate villi of the bowel lining – those same villi that are damaged by the “big 4” foods of gluten grains (wheat, barley, rye), dairy, soy and corn. Could celiac disease lead to weakened collagen and hypermobile joints? Most definitely! This is a major problem among celiac youngsters.

But what about dogs? We were taught that dogs and cats are not dependent upon dietary vitamin C because they possess the ability to make their own ascorbic acid in their liver. This is true…partly. Yes, we were told this in veterinary school but the details of how they do this were never explained to us. Vitamin C is made from glucose in a four step, enzyme-driven process taking place in the liver, that amazing organ that our colleagues practicing Eastern medicine call the center of our medical universe. They are only off by a matter of inches. Why do I boldly state that as fact? Because the liver is completely dependent upon the nutrients coming out of the small intestine, thus making the small bowel the true center of our vast biological galaxy.

Now imagine a dog or a cat being fed a grain-based diet (wheat, barley, corn, rice) laced with soy and dairy. This is the composition of numerous commercial dry kibbles. What if they suffer from a food intolerance to one or more of those proteins in the way that celiacs do to gluten? This happens much more frequently than we’ve even imagined. And, yes, they will fail to absorb the proper amounts of calcium, iron, iodine, trace minerals, proteins, fats, sugars and vitamin C just like as the gluten intolerant individual does when their villi are damaged. This can ultimately lead us into every medical condition known to man, especially when we understand essential body functions, including the immune system and enzyme-driven activities in the liver (e.g. vitamin C and protein production).

Enzyme systems are made up of proteins and cofactors, the latter being vitamins (e.g. B complex), minerals, and other enzymes. An enzyme system must have adequate cofactors to operate properly. Back to the production of vitamin C: What if the dog is not receiving adequate vitamins and minerals in their diet or is malabsorbing those that are in the diet due to food intolerance? The enzyme systems will eventually fail, including the four that are involved in the conversion of glucose to vitamin C. This is not hard to imagine. So, is the dog truly independent of dietary vitamin C as we have assumed for years? Perhaps so, if it is getting a biologically appropriate, non-offending diet that provides all of the substrates it needs to perform this vital task. (However, many nutritionists believe that the dog gets vitamin C in their natural diet to augment what they make in their liver. This would open another avenue for “subclinical scurvy” in the dog, as the food intolerant pet fails to absorb the vitamin C that is commonly added to many commercial pet foods “just in case”.)

The epiphany comes when we grasp the prenatal effects of this degenerative process. I like to remind people that we and our pets are born fully formed, just incompletely developed. Every ligament, tendon, bone and organ is in place at birth. These elements simply have to grow and mature as our bodies reach adulthood. Imagine what happens if the initial building blocks for these critical structures are inadequate or absent during the initial construction. It would be like building a house out of faulty lumbar or defective bricks or laying a subpar foundation under a two story house. The first major storm that comes along could do immeasurable harm to the structure. In fact, it could come apart at the seams.

The first evidence of failure in the poorly constructed individual is often found in the skeletal system of large dogs for the reasons mentioned above. The stress put on the frames of rapidly growing large breeds can be tremendous. But we also have certain breeds that we call chondrodysplastic, a bit of med-speak that basically means “messed up cartilage”. This is an example of genetic engineering at its worst as man created the flat-faced, squatty body dogs that make up this class- the English Bulldog, Shih Tzu, Lhasa, Bassett hound, Dachshund, and others. I love these breeds as much as the next veterinarian but our forefathers made some serious blunders in making dogs look the way they wanted them to appear, especially the first two.

The Bulldog and Shi Tzu suffer from collagen problems within the first months of life in the form of “cherry eye”, a weakness in the cartilage that gives the third eyelid shape and supports a lymph gland it contains. Their bent legs, deviated septums, congested throats, elongated palates and back problems are often overshadowed by their horrible skin allergies and ear infections, signs that the damage is being done to the intestinal lining by the “big 4” foods. I have not met many English Bulldogs or Shih Tzus that did not have major skin or ear problems…before I put them on hypoallergenic diets. They also tend to develop Demodectic mange, sign that their immune system can’t handle the mites that nearly all dogs have in their skin, passed on to them by their mother. This points directly to the malnutrition that results from the damage taking place in the center of their galaxy.

Then we have the intermediate chondrodysplastics – the Cocker, Poodle, Schnauzer, Westie, Jack Russell, Cavalier and others. These breeds often develop collapsing trachea syndrome, intervertebral Disc syndrome and heart valve problems – all collagen. Tragically, 50% of Cavies develops acute mitral valve prolapse at 5 years of age with 90% having died from congestive heart failure by age 10. Why does this particular collagen failure wait 5-10 years to show up, knowing that heart valves are flopping back and forth at the rate of 60-200 beats per minute? Interestingly, this valvular demise occurs right around the time that the Cockers are rupturing their (left) cruciate ligaments, the Dachshunds are blowing discs, the Labs are developing lower back issues, the Dobies are dying of cardiomyopathy,  and the Rottie, German Shepherd, Irish Wolfhound and Irish setter are dying from bone cancer? That’s a whole ‘nother paper but here’s a hint:  It involves the residential organisms that have taken up residence in the collagen (e.g. viruses and mycoplasma). Another clue is the spike in other “autoimmune diseases” taking place in other breeds during this time period.

The lesson to be learned is that all of these tragedies are related events. The body is one big orchestra and the conductor is the immune system. Once we start playing out of key, the conductor goes to work. We get out of tune early in life by eating the wrong things and not eating the right things. Those are two different insults. The cascade of events that follows is predictable. We just have to have an ear for the music. And, once again, this symphony starts long before our offspring are even born.

We know what we have to do to reverse this dangerous trend. It is not a requirement that we solve more mysteries or uncover more secrets in order to achieve optimal health, even after sinking into the abyss as I have done. Do we need to name every virus or bacteria that is involved in our man-made dilemma. After all, they are simply reacting (adapting) to our every insult. No, the solution is amazingly simple: We must consume biologically appropriate, non-offending foods and feed our pets in the same manner, starting with the prenatal period and continuing through childhood, adulthood, and into our twilight years. If we do this alone, medical miracles will occur.

Here’s the parting shot and the takeaway message: We can and will eliminate hip dysplasia in the dog by feeding the soon-to-be pregnant female a healthy, non-damaging diet before she is bred and throughout her gestation. Of course, the puppy diets must follow suit. We have already proven this in individuals with a history of producing dysplastic puppies. The results have been amazing, yet as predictable as rain on the horizon. I believe down to my socks that this would also be true of innumerable other collagen disorders, the likes of which are mentioned above.

We’ve all heard the expression “Garbage in, garbage out” – a perfect summation for any article that deals with the intricate functions of these incredible machines we call our body, including one written about a “genetic condition” –  hip dysplasia.

Dogtor J

Read more on collagen disorders…

More on “genetics”…


Just Desserts

These are little tidbits of knowledge I’ve discovered or insights I’ve gained over the years while doing my in-depth studies.For example, did you know researchers have found that nearly 40% of the genetic information in our DNA is viral information? This explains what we call “genetic diseases” including familial and breed tendencies toward food intolerance (e.g. celiac disease), neurological disorders (e.g. epilepsy), and numerous cancers.

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Blogtor J

Welcome to the blog of the new New research into the origin of our medical woes has revealed something startling: As it turns out, we are our own worst enemy. Yes, the Pogo quote of yesteryear found in the title of this article is quite accurate when applied to our medical lives. We love to discuss those things that we call “causes” of diseases even though we often have little clue as to how these things really cause illness. Even medical professionals can have difficulty grasping the true cause-and-effect. But that is understandable once some insight is gained into the true nature of medical training.

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News Flash!

In this section, I will be placing links to the latest breaking news in the food world, including updates on the use of elimination diets to control disease, articles on pet food, the truth about GMO foods and more. I will do my best to balance the bad with the good, but try to remember: Much of this will seem like bad news but at least we know about it now…and can change it!


With some of these stories, I will include a link to a blog entry so that you can comment on the article. This idea came to me after reading the first entry, which is a news flash that made my blood boil.

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Today’s Specials
The Simple Truth about Dog Food and Heart Disease The Epilepsy Diet Made Simple The Origin of Disease Lectins – The Missing Links Viruses – Friend or Foe? Pain, Pain Go Away
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Something to Chew On

What is the “Leaky Gut”?
The “leaky gut syndrome” is the root of many medical evils once the “big 4” induce it. Read how Italians have survived being a pasta-based society.

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Hip Dysplasia – “Genetics” vs. Diet

As misconceptions go, this is a high-priority item. Hip dysplasia is not what we were taught.

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Epilepsy and Diet

I have been studying “idiopathic epilepsy” extensively since April of 2000. Guess what? It’s not idiopathic anymore. A diet change can cure it!

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The Truth About Pet Food

Dogs and cats are carnivores yet most pet foods are grain-based. Knowing how to read a label is also very important. Does your pet food really have vegetables in it? Really???

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How to Control Epilepsy Naturally

I have been successfully treating pets with epilepsy using diet changes alone for nearly 10 years. The results have been astounding. This paper helps to summarize my findings and recommendations.

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We Have Met the Enemy and He is Us

This is the first blog placed on (6-24-07) and pretty much covers the gamut of topics – from heartburn to cancer – that are discussed on this Website. The bottom line? Viruses and bacteria are not the enemy. We are! The good news: We do have our health destinies in our own hands.

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Gluten Intolerance and Your Pet

This paper is an article I wrote for the newsletter of when asked about the prevalence of celiac disease (gluten intolerance) in the dog and cat. This condition has been definitively diagnosed in the Irish Setter but not many other breeds of dogs. I will not be at all surprised when we find that it does exist in numerous other breeds… and even the lovable mutt…but as this article explains, that may very well be a moot point. Gluten is only the beginning.

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The Answer

This 40-plus page paper is my first major paper, written in 2001 and hurriedly placed on this site on that fateful and tragic day of 9-11. It represents the culmination of nearly two years of research on the subject of food-related disorders and contains an amazing amount of facts that have been hidden from public view concerning this subject. It covers how the “big 4” trouble foods- gluten, dairy, soy and corn- came into being, catapulted into common usage, and became directly involved in most of our serious medical conditions, including epilepsy, chronic fatigue, irritable bowel syndrome, insomnia, ADHD, pain syndromes, depression, and allergies. Severe immune-mediated diseases such as diabetes, lupus, and rheumatoid arthritis are also covered.


Yes, it is quite long, but it appeared on my computer screen and was placed onto the Web just as it came out of my head. And it is wordy in places. But, as I tell my clients, “There is no law that you have to read it all in one sitting. Think of it as a free, short book rather than a long research paper. It’s all how you look at it, right?”


The one thing I can say is that this information WILL change your life…guaranteed!!!

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